Acessibilidade / Reportar erro

Case 2/2008: twenty two year old male with ventricular septal defect of persistently small proportions

CLINICORADIOLOGICAL SESSION

Case 2/2008 – Twenty two year old male with ventricular septal defect of persistently small proportions

Edmar Atik

Clínica do Dr. Edmar Atik - São Paulo, SP - Brazil

Mailing address Mailing address: Edmar Atik InCor Av. Dr. Enéas Carvalho de Aguiar, 44 05403-000 - São Paulo, SP - Brazil E-mail: conatik@incor.usp.br

Key words: Ventricular septal defect, congenital cardiopathy, clinical.

Clinical data

Cardiac murmur with early detection in the first days of life which remained unchanged over time. During this period, the patient experienced no symptoms or clinical complications and was able to participate normally in routine physical activities.

Physical examination

Patient was eupneic, had good skin color, and pulses were palpated on all 4 limbs. Weight: 64 Kg, Height: 170cm, BP: 125/70 mm Hg, HR: 76 bpm. Aorta was not palpated at the supraesternal notch. Precordium showed no deformities or impulsions. Ictus cordis was not palpated. Normal heart sounds, with inconsistent splitting of second heart sound. Rough ejection systolic murmur +/++ in 3rd, 2nd, and 4th left intercostal spaces, not accompanied by thrills.

Lungs and abdomen: no abnormalities.

The electrocardiogram showed normal sinus rhythm and normal electrical potentials. ÂP: + 50º, ÂQRS: +70º, ÂT: +50º (Fig. 1).


Radiographic image

Cardiac area shows normal dimensions and slightly increased vascular network (rete), primarily in the hilar region (Fig. 2).


Diagnostic impression

This image is consistent with a slight shift of blood flow from left to right as in congenital acyanogenic cardiopathies such as ventricular septal defect.

Differential diagnosis

Other cardiopathies should be considered, such as ASD (atrial septal defect), PDA, and AV fistulas, as long as they cause minor repercussions.

Diagnostic confirmations

Clinical elements easily point towards the diagnosis of ventricular septal defect with mild hemodynamic repercussions. From the first days of life and to date, the echocardiogram has shown outlet muscular VSD (ventricular septal defect) with 2 to 3 mm diameter and normal cardiac chambers (Fig. 3).


Treatment

Always considered conservative, with close clinical observation in light of the mild clinical and laboratorial manifestations and following rigid prophylactic orientation for infectious endocarditis.

The same orientation continued to be adopted because of the perspective of life expectancy similar to that of the normal population.

  • Mailing address:
    Edmar Atik
    InCor
    Av. Dr. Enéas Carvalho de Aguiar, 44
    05403-000 - São Paulo, SP - Brazil
    E-mail:
  • Publication Dates

    • Publication in this collection
      29 May 2008
    • Date of issue
      Apr 2008
    Sociedade Brasileira de Cardiologia - SBC Avenida Marechal Câmara, 160, sala: 330, Centro, CEP: 20020-907, (21) 3478-2700 - Rio de Janeiro - RJ - Brazil, Fax: +55 21 3478-2770 - São Paulo - SP - Brazil
    E-mail: revista@cardiol.br